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Let us now take the half life of popular anabolic steroids and their derivatives into the account, and examine the relationship amongst the effects of anabolic steroids in the brain. We know that the half life of anabolic steroids is from the time when they are administered to the body, for which they may have a maximum systemic distribution of at least 6 h. In contrast to these half-lives, the short half-life of other neurotransmitters like serotonin (50–65 h)  and dopamine (7–15 h) , for example, can be a consequence of receptor sensitivity to their action and can be prolonged by exogenous administration of anabolic steroids  or other physiological agents, especially during stress , using ostarine during pct. While the half-life of anabolic androgenic steroids is short by comparison with other compounds, we also know that their effects on the developing and adult brain of male rat brains are of much lower magnitude, andarine s4 fat loss. The average half-life of aldosterone (4, dbol half life.0 weeks) and testosterone (2, dbol half life.6 days) is a consequence of the relatively long half-life of these steroid hormones, as they can be absorbed from adipose tissue and used to make their metabolites within the blood plasma after their metabolism in the liver , dbol half life. By contrast, the anabolic androgenic steroids and their derivatives have a relatively short half-time (<200 h in many rats). These steroids have been found to affect brain development and adult behaviour in numerous studies; for example, in animal models of aggression, stress, and anxiety , . We now know that the half-life of anabolic-androgenic steroids also depends on their body distribution, hgh buy canada. Anabolic- and anandamide-containing steroids such as testosterone, DHT, theanine, and some steroid derivatives such as androestrogens and dihydro-testosterone, are metabolized predominantly by the liver while anandamide is metabolized mainly by the adrenal glands and to a lesser degree by the liver . While the metabolic pathway of theanine remains largely unknown, the presence of both anandamide and anandamide in liver biotransformation of various anabolic- and anandamide-containing steroids suggests that they could not only be produced centrally and eliminated after degradation in the liver, but also could be transported to the brain to act on the dopaminergic receptors, winsol brakel.
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